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Furthermore, the physiological or therapeutic significance of this enzymatic cleavage is unclear because DPP4 inhibitors universally decrease levels of intact glucagon in rodents and humans via potentiation of GLP-1 action, and circulating levels of glucagon [3–29] and glucagon [5–29] have not been reported in animals or humans treated with a DPP4 inhibitor

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GLP-1 RAs may inhibit retinal neurodegeneration and reduce proinflammatory processes, which could protect against diabetic retinopathy progression